EVIDENCE OF INCREASED ENDOGENOUS CARBON MONOXIDE PRODUCTION IN NEWBORN RAT ENDOTOXICOSIS

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EVIDENCE OF INCREASED ENDOGENOUS CARBON MONOXIDE PRODUCTION IN NEWBORN RAT ENDOTOXICOSIS

中国医学科学杂志（英文版） 1997年0卷第4期页码：212-215

Updated：2024-04-10

- EVIDENCE OF INCREASED ENDOGENOUS CARBON MONOXIDE PRODUCTION IN NEWBORN RAT ENDOTOXICOSIS
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  中图分类号： R722.1
- 纸质出版日期：1997
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EVIDENCE OF INCREASED ENDOGENOUS CARBON MONOXIDE PRODUCTION IN NEWBORN RAT ENDOTOXICOSIS[J]. 中国医学科学杂志（英文版）, 1997,0(4):212-215.

EVIDENCE OF INCREASED ENDOGENOUS CARBON MONOXIDE PRODUCTION IN NEWBORN RAT ENDOTOXICOSIS[J]. Chinese medical sciences journal, 1997, 0(4): 212-215.
EVIDENCE OF INCREASED ENDOGENOUS CARBON MONOXIDE PRODUCTION IN NEWBORN RAT ENDOTOXICOSIS[J]. 中国医学科学杂志（英文版）, 1997,0(4):212-215. DOI：

EVIDENCE OF INCREASED ENDOGENOUS CARBON MONOXIDE PRODUCTION IN NEWBORN RAT ENDOTOXICOSIS[J]. Chinese medical sciences journal, 1997, 0(4): 212-215. DOI：

摘要

Abstract

Carbon monoxide is thought to serve as a new endogenous mediator in the pathogenesis of sepsis andseptic shock. In newborn rat endotoxicosis

carbon monoxide levels in the circulation as well as liver

kid-ney and lung were found to be significantly increased （P<0. 05）. Moreover

the elevations of carbonmonoxide correlated with enhanced nitric oxide production as indicated by nitrite/nitrate levels （P<0. 05）.Our present data showed for the first time that endogenously produced carbon monoxide was increasedduring the course of shock-like states

which suggested that the role of carbon monoxide in sepsis and sep-tic shock might worth further study.

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ROLE OF ENDOGENOUS CARBON MONOXIDE IN ENDOTOXIN SHOCK